Epistatic interactions: how strong in disease and evolution?

Azevedo, Luísa

Epistatic interactions: how strong in disease and evolution? [electronic resource] - Trends in genetics : TIG Nov 2006 - 581-5 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Review

0168-9525

10.1016/j.tig.2006.08.001 doi


Adaptation, Biological
Adaptor Proteins, Signal Transducing
Alleles
Animals
Biological Evolution
Carrier Proteins--genetics
Cytoskeletal Proteins--genetics
Dosage Compensation, Genetic
Epistasis, Genetic
Eye Proteins--genetics
Genetic Diseases, Inborn--genetics
Glycoproteins--genetics
Group II Chaperonins
Humans
Models, Molecular
Molecular Chaperones--genetics
MutL Protein Homolog 1
Mutation
Nuclear Proteins--genetics
Pan troglodytes--genetics
Recombination, Genetic
Transcription Factors--genetics
Trypsin
Trypsinogen--genetics
AIRE Protein