Overexpression of inducible heat shock protein 70 and its mutants in astrocytes is associated with maintenance of mitochondrial physiology during glucose deprivation stress.

Ouyang, Yi-Bing

Overexpression of inducible heat shock protein 70 and its mutants in astrocytes is associated with maintenance of mitochondrial physiology during glucose deprivation stress. [electronic resource] - Cell stress & chaperones 2006 - 180-6 p. digital

Publication Type: Journal Article; Research Support, N.I.H., Extramural

1355-8145

10.1379/csc-182r.1 doi


Animals
Animals, Newborn
Astrocytes--cytology
Cell Respiration--drug effects
Cells, Cultured
Glucose--deficiency
HSP70 Heat-Shock Proteins--biosynthesis
Humans
Mice
Mitochondria--drug effects
Mutagenesis, Site-Directed
Mutant Proteins--biosynthesis
Reactive Oxygen Species--metabolism