The deficiency of Akt1 is sufficient to suppress tumor development in Pten+/- mice.
Chen, Mei-Ling
The deficiency of Akt1 is sufficient to suppress tumor development in Pten+/- mice. [electronic resource] - Genes & development Jun 2006 - 1569-74 p. digital
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
0890-9369
10.1101/gad.1395006 doi
Adrenal Gland Neoplasms--enzymology
Animals
Endometrial Neoplasms--enzymology
Female
Heterozygote
Intestinal Polyps--enzymology
Male
Mice
Neoplasms--enzymology
PTEN Phosphohydrolase--genetics
Prostatic Intraepithelial Neoplasia--enzymology
Prostatic Neoplasms--enzymology
Proto-Oncogene Proteins c-akt--deficiency
Pseudolymphoma--enzymology
Thyroid Neoplasms--enzymology
The deficiency of Akt1 is sufficient to suppress tumor development in Pten+/- mice. [electronic resource] - Genes & development Jun 2006 - 1569-74 p. digital
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
0890-9369
10.1101/gad.1395006 doi
Adrenal Gland Neoplasms--enzymology
Animals
Endometrial Neoplasms--enzymology
Female
Heterozygote
Intestinal Polyps--enzymology
Male
Mice
Neoplasms--enzymology
PTEN Phosphohydrolase--genetics
Prostatic Intraepithelial Neoplasia--enzymology
Prostatic Neoplasms--enzymology
Proto-Oncogene Proteins c-akt--deficiency
Pseudolymphoma--enzymology
Thyroid Neoplasms--enzymology