CV-11974, angiotensin II type I receptor antagonist, protects against ischemia-reperfusion injury of the small intestine in rats.
Takagi, Tomohisa
CV-11974, angiotensin II type I receptor antagonist, protects against ischemia-reperfusion injury of the small intestine in rats. [electronic resource] - European journal of pharmacology Mar 2006 - 283-90 p. digital
Publication Type: Journal Article
0014-2999
10.1016/j.ejphar.2006.02.005 doi
Angiotensin II Type 1 Receptor Blockers--pharmacology
Animals
Benzimidazoles--pharmacology
Biphenyl Compounds
Cell Adhesion--drug effects
Cell Line
Chemokine CXCL1
Chemokines, CXC--genetics
Dose-Response Relationship, Drug
Endothelial Cells--cytology
Gene Expression--drug effects
Hemoglobins--metabolism
Humans
Intestinal Diseases--etiology
Intestinal Mucosa--drug effects
Intestine, Small--drug effects
Male
Neutrophils--cytology
Peroxidase--metabolism
Proteins--metabolism
RNA, Messenger--genetics
Rats
Rats, Wistar
Reperfusion Injury--complications
Reverse Transcriptase Polymerase Chain Reaction
Tetrazoles--pharmacology
Thiobarbituric Acid Reactive Substances--metabolism
CV-11974, angiotensin II type I receptor antagonist, protects against ischemia-reperfusion injury of the small intestine in rats. [electronic resource] - European journal of pharmacology Mar 2006 - 283-90 p. digital
Publication Type: Journal Article
0014-2999
10.1016/j.ejphar.2006.02.005 doi
Angiotensin II Type 1 Receptor Blockers--pharmacology
Animals
Benzimidazoles--pharmacology
Biphenyl Compounds
Cell Adhesion--drug effects
Cell Line
Chemokine CXCL1
Chemokines, CXC--genetics
Dose-Response Relationship, Drug
Endothelial Cells--cytology
Gene Expression--drug effects
Hemoglobins--metabolism
Humans
Intestinal Diseases--etiology
Intestinal Mucosa--drug effects
Intestine, Small--drug effects
Male
Neutrophils--cytology
Peroxidase--metabolism
Proteins--metabolism
RNA, Messenger--genetics
Rats
Rats, Wistar
Reperfusion Injury--complications
Reverse Transcriptase Polymerase Chain Reaction
Tetrazoles--pharmacology
Thiobarbituric Acid Reactive Substances--metabolism