Pharmacologic evidence for abnormal thalamocortical functioning in GABA receptor beta3 subunit-deficient mice, a model of Angelman syndrome.

Handforth, Adrian

Pharmacologic evidence for abnormal thalamocortical functioning in GABA receptor beta3 subunit-deficient mice, a model of Angelman syndrome. [electronic resource] - Epilepsia Dec 2005 - 1860-70 p. digital

Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.

0013-9580

10.1111/j.1528-1167.2005.00287.x doi


Angelman Syndrome--drug therapy
Animals
Anticonvulsants--pharmacology
Baclofen--pharmacology
Cerebral Cortex--drug effects
Disease Models, Animal
Electrodes, Implanted
Electroencephalography--drug effects
Ethosuximide--pharmacology
Female
GABA Agonists--pharmacology
GABA Antagonists--pharmacology
Humans
Male
Mice
Mice, Mutant Strains
Neural Pathways--physiopathology
Organophosphorus Compounds--pharmacology
Parietal Lobe--physiopathology
Receptors, GABA-A--deficiency
Thalamus--drug effects