Endothelial nitric oxide synthase activation by tumor necrosis factor alpha through neutral sphingomyelinase 2, sphingosine kinase 1, and sphingosine 1 phosphate receptors: a novel pathway relevant to the pathophysiology of endothelium.

De Palma, Clara

Endothelial nitric oxide synthase activation by tumor necrosis factor alpha through neutral sphingomyelinase 2, sphingosine kinase 1, and sphingosine 1 phosphate receptors: a novel pathway relevant to the pathophysiology of endothelium. [electronic resource] - Arteriosclerosis, thrombosis, and vascular biology Jan 2006 - 99-105 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

1524-4636

10.1161/01.ATV.0000194074.59584.42 doi


Calcium--metabolism
Cell Adhesion--drug effects
Cells, Cultured
Endothelium, Vascular--cytology
Enzyme Activation--drug effects
Humans
Nitric Oxide Synthase Type III--metabolism
Phosphatidylinositol 3-Kinases--metabolism
Phosphotransferases (Alcohol Group Acceptor)--metabolism
Proto-Oncogene Proteins c-akt--metabolism
Receptors, Lysosphingolipid--metabolism
Signal Transduction--drug effects
Sphingomyelin Phosphodiesterase--metabolism
Tumor Necrosis Factor-alpha--metabolism
Umbilical Veins--cytology