Multiple signals induce endoplasmic reticulum stress in both primary and immortalized chondrocytes resulting in loss of differentiation, impaired cell growth, and apoptosis.

Yang, Ling

Multiple signals induce endoplasmic reticulum stress in both primary and immortalized chondrocytes resulting in loss of differentiation, impaired cell growth, and apoptosis. [electronic resource] - The Journal of biological chemistry Sep 2005 - 31156-65 p. digital

Publication Type: Journal Article

0021-9258

10.1074/jbc.M501069200 doi


Animals
Annexin A5--metabolism
Anti-Bacterial Agents--metabolism
Apoptosis--physiology
Biomarkers
CCAAT-Enhancer-Binding Proteins--metabolism
Caspase 12
Caspases--metabolism
Cell Differentiation--physiology
Cells, Cultured
Chondrocytes--cytology
Collagen Type II--metabolism
DNA Fragmentation
Endoplasmic Reticulum--metabolism
Extracellular Matrix--metabolism
Gene Expression Regulation
Glucose--metabolism
Proliferating Cell Nuclear Antigen--metabolism
Rats
Signal Transduction--physiology
Thapsigargin--metabolism
Transcription Factor CHOP
Transcription Factors--metabolism
Tunicamycin--metabolism