N-terminal tripeptide of IGF-1 improves functional deficits after 6-OHDA lesion in rats.
Krishnamurthi, Rita
N-terminal tripeptide of IGF-1 improves functional deficits after 6-OHDA lesion in rats. [electronic resource] - Neuroreport Jul 2004 - 1601-4 p. digital
Publication Type: Comparative Study; Journal Article
0959-4965
10.1097/01.wnr.0000127461.15985.07 doi
Animals
Behavior, Animal--drug effects
Cell Count--methods
Corpus Striatum--injuries
Disease Models, Animal
Dose-Response Relationship, Drug
Functional Laterality
Immunohistochemistry--methods
Insulin-Like Growth Factor I--therapeutic use
Male
Motor Activity--drug effects
Neuroprotective Agents--therapeutic use
Oligopeptides--therapeutic use
Oxidopamine--toxicity
Parkinson Disease--drug therapy
Psychomotor Performance--drug effects
Rats
Rats, Wistar
Rotation
Substantia Nigra--injuries
Sympatholytics--toxicity
Time Factors
Tyrosine 3-Monooxygenase--metabolism
N-terminal tripeptide of IGF-1 improves functional deficits after 6-OHDA lesion in rats. [electronic resource] - Neuroreport Jul 2004 - 1601-4 p. digital
Publication Type: Comparative Study; Journal Article
0959-4965
10.1097/01.wnr.0000127461.15985.07 doi
Animals
Behavior, Animal--drug effects
Cell Count--methods
Corpus Striatum--injuries
Disease Models, Animal
Dose-Response Relationship, Drug
Functional Laterality
Immunohistochemistry--methods
Insulin-Like Growth Factor I--therapeutic use
Male
Motor Activity--drug effects
Neuroprotective Agents--therapeutic use
Oligopeptides--therapeutic use
Oxidopamine--toxicity
Parkinson Disease--drug therapy
Psychomotor Performance--drug effects
Rats
Rats, Wistar
Rotation
Substantia Nigra--injuries
Sympatholytics--toxicity
Time Factors
Tyrosine 3-Monooxygenase--metabolism