Inactivation of p16INK4a by CpG hypermethylation is not a frequent event in colorectal cancer.
Norrie, Mark W A
Inactivation of p16INK4a by CpG hypermethylation is not a frequent event in colorectal cancer. [electronic resource] - Journal of surgical oncology Nov 2003 - 143-50 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
0022-4790
10.1002/jso.10310 doi
Colorectal Neoplasms--genetics
CpG Islands
Cyclin-Dependent Kinase Inhibitor p16--biosynthesis
DNA Methylation
DNA, Neoplasm--genetics
Female
Gene Silencing
Genes, p16
Genes, ras
Humans
Immunohistochemistry
Loss of Heterozygosity
Male
Mutation
Polymerase Chain Reaction
Promoter Regions, Genetic
Inactivation of p16INK4a by CpG hypermethylation is not a frequent event in colorectal cancer. [electronic resource] - Journal of surgical oncology Nov 2003 - 143-50 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
0022-4790
10.1002/jso.10310 doi
Colorectal Neoplasms--genetics
CpG Islands
Cyclin-Dependent Kinase Inhibitor p16--biosynthesis
DNA Methylation
DNA, Neoplasm--genetics
Female
Gene Silencing
Genes, p16
Genes, ras
Humans
Immunohistochemistry
Loss of Heterozygosity
Male
Mutation
Polymerase Chain Reaction
Promoter Regions, Genetic