Calcium influx evoked by Ca2+ store depletion in human platelets is more susceptible to cytochrome P-450 inhibitors than receptor-mediated calcium entry.

Sargeant, P

Calcium influx evoked by Ca2+ store depletion in human platelets is more susceptible to cytochrome P-450 inhibitors than receptor-mediated calcium entry. [electronic resource] - Cell calcium Oct 1992 - 553-64 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

0143-4160

10.1016/0143-4160(92)90035-q doi


Adenosine Diphosphate--pharmacology
Blood Platelets--drug effects
Calcium--metabolism
Calcium Channels--drug effects
Calcium-Transporting ATPases--antagonists & inhibitors
Cell Compartmentation
Cytochrome P-450 Enzyme Inhibitors
Cytochrome P-450 Enzyme System--physiology
Econazole--pharmacology
Humans
Hydroquinones--pharmacology
Inositol 1,4,5-Trisphosphate--physiology
Intracellular Fluid--metabolism
Ion Channel Gating--drug effects
Manganese--metabolism
Miconazole--pharmacology
Osmolar Concentration
Platelet Activating Factor--pharmacology
Platelet Activation--drug effects
Receptors, Cholinergic--physiology
Ryanodine Receptor Calcium Release Channel
Signal Transduction
Terpenes--pharmacology
Thapsigargin
Thrombin--pharmacology
Vasopressins--pharmacology