Time course of early motor and neuropathological anomalies in a knock-in mouse model of Huntington's disease with 140 CAG repeats.
Menalled, Liliana B
Time course of early motor and neuropathological anomalies in a knock-in mouse model of Huntington's disease with 140 CAG repeats. [electronic resource] - The Journal of comparative neurology Oct 2003 - 11-26 p. digital
Publication Type: Journal Article
0021-9967
10.1002/cne.10776 doi
Animals
Brain--metabolism
Cerebral Cortex--metabolism
Corpus Striatum--metabolism
Disease Models, Animal
Female
Grooming--physiology
Huntingtin Protein
Huntington Disease--genetics
Immunohistochemistry
Inclusion Bodies--genetics
Lameness, Animal--genetics
Male
Mice
Mice, Transgenic
Motor Activity--genetics
Nerve Tissue Proteins--deficiency
Neurons--metabolism
Neuropil--metabolism
Nuclear Proteins--deficiency
Nucleus Accumbens--metabolism
Olfactory Pathways--metabolism
Recombinant Fusion Proteins--genetics
Time Factors
Trinucleotide Repeats--genetics
Time course of early motor and neuropathological anomalies in a knock-in mouse model of Huntington's disease with 140 CAG repeats. [electronic resource] - The Journal of comparative neurology Oct 2003 - 11-26 p. digital
Publication Type: Journal Article
0021-9967
10.1002/cne.10776 doi
Animals
Brain--metabolism
Cerebral Cortex--metabolism
Corpus Striatum--metabolism
Disease Models, Animal
Female
Grooming--physiology
Huntingtin Protein
Huntington Disease--genetics
Immunohistochemistry
Inclusion Bodies--genetics
Lameness, Animal--genetics
Male
Mice
Mice, Transgenic
Motor Activity--genetics
Nerve Tissue Proteins--deficiency
Neurons--metabolism
Neuropil--metabolism
Nuclear Proteins--deficiency
Nucleus Accumbens--metabolism
Olfactory Pathways--metabolism
Recombinant Fusion Proteins--genetics
Time Factors
Trinucleotide Repeats--genetics