The JAK/STAT pathway is not sufficient to sustain the antiproliferative response in an interferon-resistant human melanoma cell line.
Jackson, David P
The JAK/STAT pathway is not sufficient to sustain the antiproliferative response in an interferon-resistant human melanoma cell line. [electronic resource] - Melanoma research Jun 2003 - 219-29 p. digital
Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
0960-8931
10.1097/00008390-200306000-00001 doi
Antineoplastic Agents--pharmacology
Cell Division--drug effects
DNA-Binding Proteins--drug effects
Drug Resistance, Neoplasm--physiology
Humans
Interferons--pharmacology
Janus Kinase 1
Janus Kinase 2
Janus Kinase 3
Melanoma--drug therapy
Protein-Tyrosine Kinases--metabolism
Proto-Oncogene Proteins
STAT1 Transcription Factor
STAT2 Transcription Factor
STAT3 Transcription Factor
Signal Transduction
Skin Neoplasms--drug therapy
Trans-Activators--drug effects
Tumor Cells, Cultured
The JAK/STAT pathway is not sufficient to sustain the antiproliferative response in an interferon-resistant human melanoma cell line. [electronic resource] - Melanoma research Jun 2003 - 219-29 p. digital
Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
0960-8931
10.1097/00008390-200306000-00001 doi
Antineoplastic Agents--pharmacology
Cell Division--drug effects
DNA-Binding Proteins--drug effects
Drug Resistance, Neoplasm--physiology
Humans
Interferons--pharmacology
Janus Kinase 1
Janus Kinase 2
Janus Kinase 3
Melanoma--drug therapy
Protein-Tyrosine Kinases--metabolism
Proto-Oncogene Proteins
STAT1 Transcription Factor
STAT2 Transcription Factor
STAT3 Transcription Factor
Signal Transduction
Skin Neoplasms--drug therapy
Trans-Activators--drug effects
Tumor Cells, Cultured