Boswellic acids trigger apoptosis via a pathway dependent on caspase-8 activation but independent on Fas/Fas ligand interaction in colon cancer HT-29 cells.

Liu, Jian-Jun

Boswellic acids trigger apoptosis via a pathway dependent on caspase-8 activation but independent on Fas/Fas ligand interaction in colon cancer HT-29 cells. [electronic resource] - Carcinogenesis Dec 2002 - 2087-93 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

0143-3334

10.1093/carcin/23.12.2087 doi


Anti-Inflammatory Agents--pharmacology
Apoptosis
Blotting, Western
Boswellia--metabolism
Camptothecin--pharmacology
Caspase 3
Caspase 8
Caspase 9
Caspases--metabolism
Cell Division
Cell Line
Cell Survival
Cytoplasm--metabolism
DNA--biosynthesis
Dose-Response Relationship, Drug
Electrophoresis, Polyacrylamide Gel
Enzyme Activation
Enzyme Inhibitors--pharmacology
Enzyme-Linked Immunosorbent Assay
Fas Ligand Protein
Flow Cytometry
Histones--metabolism
Humans
Membrane Glycoproteins--metabolism
Poly(ADP-ribose) Polymerases--metabolism
Protein Binding
Signal Transduction
Time Factors
Triterpenes--pharmacology
Tumor Cells, Cultured
fas Receptor--metabolism