Atherosclerotic lesions and mitochondria DNA deletions in brain microvessels as a central target for the development of human AD and AD-like pathology in aged transgenic mice.
Aliev, Gjumrakch
Atherosclerotic lesions and mitochondria DNA deletions in brain microvessels as a central target for the development of human AD and AD-like pathology in aged transgenic mice. [electronic resource] - Annals of the New York Academy of Sciences Nov 2002 - 45-64 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
0077-8923
10.1111/j.1749-6632.2002.tb04798.x doi
Alzheimer Disease--genetics
Amyloid beta-Protein Precursor--genetics
Animals
Arteriosclerosis--pathology
Base Sequence
Cerebrovascular Disorders--pathology
DNA Probes
DNA, Mitochondrial--genetics
Humans
Mice
Mice, Transgenic
Microcirculation--pathology
Models, Animal
Oxidative Stress
Sequence Deletion
Atherosclerotic lesions and mitochondria DNA deletions in brain microvessels as a central target for the development of human AD and AD-like pathology in aged transgenic mice. [electronic resource] - Annals of the New York Academy of Sciences Nov 2002 - 45-64 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
0077-8923
10.1111/j.1749-6632.2002.tb04798.x doi
Alzheimer Disease--genetics
Amyloid beta-Protein Precursor--genetics
Animals
Arteriosclerosis--pathology
Base Sequence
Cerebrovascular Disorders--pathology
DNA Probes
DNA, Mitochondrial--genetics
Humans
Mice
Mice, Transgenic
Microcirculation--pathology
Models, Animal
Oxidative Stress
Sequence Deletion