Prevention of cytokine-induced apoptosis by insulin-like growth factor-I is independent of cell adhesion molecules in HT29-D4 colon carcinoma cells-evidence for a NF-kappaB-dependent survival mechanism.

Garrouste, F

Prevention of cytokine-induced apoptosis by insulin-like growth factor-I is independent of cell adhesion molecules in HT29-D4 colon carcinoma cells-evidence for a NF-kappaB-dependent survival mechanism. [electronic resource] - Cell death and differentiation Jul 2002 - 768-79 p. digital

Publication Type: Journal Article

1350-9047

10.1038/sj.cdd.4401022 doi


Apoptosis
Cell Adhesion Molecules--metabolism
Cell Communication
Cell Survival
Drug Resistance
Extracellular Matrix--metabolism
Focal Adhesion Kinase 1
Focal Adhesion Protein-Tyrosine Kinases
HT29 Cells
Humans
Insulin--pharmacology
Insulin Receptor Substrate Proteins
Insulin-Like Growth Factor I--metabolism
Interferon-gamma--pharmacology
Interleukin-8--biosynthesis
Mitogen-Activated Protein Kinase 1--metabolism
Mitogen-Activated Protein Kinase 3
Mitogen-Activated Protein Kinases--metabolism
NF-kappa B--metabolism
Phosphatidylinositol 3-Kinases--metabolism
Phosphoproteins--metabolism
Phosphorylation
Protein Serine-Threonine Kinases
Protein-Tyrosine Kinases--metabolism
Proto-Oncogene Proteins--metabolism
Proto-Oncogene Proteins c-akt
Signal Transduction
Tumor Necrosis Factor-alpha--pharmacology
p38 Mitogen-Activated Protein Kinases