Blocking chemokine responsive to gamma-2/interferon (IFN)-gamma inducible protein and monokine induced by IFN-gamma activity in vivo reduces the pathogenetic but not the antiviral potential of hepatitis B virus-specific cytotoxic T lymphocytes.
Kakimi, K
Blocking chemokine responsive to gamma-2/interferon (IFN)-gamma inducible protein and monokine induced by IFN-gamma activity in vivo reduces the pathogenetic but not the antiviral potential of hepatitis B virus-specific cytotoxic T lymphocytes. [electronic resource] - The Journal of experimental medicine Dec 2001 - 1755-66 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
0022-1007
10.1084/jem.194.12.1755 doi
Animals
Chemokine CXCL10
Cytotoxicity, Immunologic
Hepatitis B--genetics
Hepatitis B virus--immunology
Interferon-gamma--genetics
Liver--immunology
Mice
Mice, Transgenic
Monokines--genetics
T-Lymphocytes, Cytotoxic--immunology
Blocking chemokine responsive to gamma-2/interferon (IFN)-gamma inducible protein and monokine induced by IFN-gamma activity in vivo reduces the pathogenetic but not the antiviral potential of hepatitis B virus-specific cytotoxic T lymphocytes. [electronic resource] - The Journal of experimental medicine Dec 2001 - 1755-66 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
0022-1007
10.1084/jem.194.12.1755 doi
Animals
Chemokine CXCL10
Cytotoxicity, Immunologic
Hepatitis B--genetics
Hepatitis B virus--immunology
Interferon-gamma--genetics
Liver--immunology
Mice
Mice, Transgenic
Monokines--genetics
T-Lymphocytes, Cytotoxic--immunology