Activation of Valpha14(+) natural killer T cells by alpha-galactosylceramide results in development of Th1 response and local host resistance in mice infected with Cryptococcus neoformans.
Kawakami, K
Activation of Valpha14(+) natural killer T cells by alpha-galactosylceramide results in development of Th1 response and local host resistance in mice infected with Cryptococcus neoformans. [electronic resource] - Infection and immunity Jan 2001 - 213-20 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
0019-9567
10.1128/IAI.69.1.213-220.2001 doi
Animals
CD4 Antigens--physiology
Cryptococcosis--immunology
Galactosylceramides--pharmacology
Interferon-gamma--biosynthesis
Interleukin-4--biosynthesis
Killer Cells, Natural--immunology
Lung--microbiology
Lymphocyte Activation
Mice
Mice, Inbred C57BL
Mice, Knockout
Spleen--microbiology
Th1 Cells--immunology
Activation of Valpha14(+) natural killer T cells by alpha-galactosylceramide results in development of Th1 response and local host resistance in mice infected with Cryptococcus neoformans. [electronic resource] - Infection and immunity Jan 2001 - 213-20 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
0019-9567
10.1128/IAI.69.1.213-220.2001 doi
Animals
CD4 Antigens--physiology
Cryptococcosis--immunology
Galactosylceramides--pharmacology
Interferon-gamma--biosynthesis
Interleukin-4--biosynthesis
Killer Cells, Natural--immunology
Lung--microbiology
Lymphocyte Activation
Mice
Mice, Inbred C57BL
Mice, Knockout
Spleen--microbiology
Th1 Cells--immunology