[An increased proinflammatory activity is inherent in insulin resistance].
Fernández-Real, J M
[An increased proinflammatory activity is inherent in insulin resistance]. [electronic resource] - Medicina clinica Jul 2000 - 185-9 p. digital
Publication Type: Journal Article; Review
0025-7753
10.1016/s0025-7753(00)71502-8 doi
Adipocytes--physiology
Adipose Tissue--cytology
Adult
Animals
Carbohydrate Metabolism
Cells, Cultured
Diabetes Mellitus, Experimental--metabolism
Diabetes Mellitus, Type 2--genetics
Female
Gene Expression
Genotype
Humans
Inflammation--etiology
Inflammation Mediators--physiology
Insulin--physiology
Insulin Resistance--genetics
Interleukin-6--genetics
Islets of Langerhans--physiopathology
Lipid Metabolism
Liver--physiopathology
Male
Mice
Mice, Knockout
Mice, Obese
Mutation
Obesity--genetics
Polymorphism, Genetic
Tumor Necrosis Factor-alpha--genetics
[An increased proinflammatory activity is inherent in insulin resistance]. [electronic resource] - Medicina clinica Jul 2000 - 185-9 p. digital
Publication Type: Journal Article; Review
0025-7753
10.1016/s0025-7753(00)71502-8 doi
Adipocytes--physiology
Adipose Tissue--cytology
Adult
Animals
Carbohydrate Metabolism
Cells, Cultured
Diabetes Mellitus, Experimental--metabolism
Diabetes Mellitus, Type 2--genetics
Female
Gene Expression
Genotype
Humans
Inflammation--etiology
Inflammation Mediators--physiology
Insulin--physiology
Insulin Resistance--genetics
Interleukin-6--genetics
Islets of Langerhans--physiopathology
Lipid Metabolism
Liver--physiopathology
Male
Mice
Mice, Knockout
Mice, Obese
Mutation
Obesity--genetics
Polymorphism, Genetic
Tumor Necrosis Factor-alpha--genetics