Surviving granule cells of the sclerotic human hippocampus have reduced Ca(2+) influx because of a loss of calbindin-D(28k) in temporal lobe epilepsy.

Nägerl, U V

Surviving granule cells of the sclerotic human hippocampus have reduced Ca(2+) influx because of a loss of calbindin-D(28k) in temporal lobe epilepsy. [electronic resource] - The Journal of neuroscience : the official journal of the Society for Neuroscience Mar 2000 - 1831-6 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.

1529-2401


Action Potentials--physiology
Adult
Calbindins
Calcium--metabolism
Calcium Channels--physiology
Dentate Gyrus--pathology
Epilepsy, Temporal Lobe--metabolism
Female
Humans
In Vitro Techniques
Male
Middle Aged
Neurons--chemistry
Patch-Clamp Techniques
S100 Calcium Binding Protein G--analysis
Sclerosis