Receptor for advanced glycation end products mediates sepsis-triggered amyloid-β accumulation, Tau phosphorylation, and cognitive impairment.

Receptor for advanced glycation end products mediates sepsis-triggered amyloid-β accumulation, Tau phosphorylation, and cognitive impairment. — المؤلف: Gasparotto, Juciano — The Journal of biological chemistry 20181127

المؤلف
Gasparotto, Juciano
النشر
The Journal of biological chemistry 20181127
الوصف
226-244 p.
الموضوعات
Amyloid beta-Peptides؛ Animals؛ Brain؛ Cognition؛ Cognitive Dysfunction؛ Glycation End Products, Advanced؛ Hippocampus؛ Inflammation