Receptor for advanced glycation end products mediates sepsis-triggered amyloid-β accumulation, Tau phosphorylation, and cognitive impairment.
Receptor for advanced glycation end products mediates sepsis-triggered amyloid-β accumulation, Tau phosphorylation, and cognitive impairment. — المؤلف: Gasparotto, Juciano — The Journal of biological chemistry 20181127
- المؤلف
- Gasparotto, Juciano
- النشر
- The Journal of biological chemistry 20181127
- الوصف
- 226-244 p.
- الموضوعات
- Amyloid beta-Peptides؛ Animals؛ Brain؛ Cognition؛ Cognitive Dysfunction؛ Glycation End Products, Advanced؛ Hippocampus؛ Inflammation