Human SOD1 ALS Mutations in a Drosophila Knock-In Model Cause Severe Phenotypes and Reveal Dosage-Sensitive Gain- and Loss-of-Function Components.
Human SOD1 ALS Mutations in a Drosophila Knock-In Model Cause Severe Phenotypes and Reveal Dosage-Sensitive Gain- and Loss-of-Function Components. — المؤلف: Şahin, Aslı — Genetics 20170530
- المؤلف
- Şahin, Aslı
- النشر
- Genetics 20170530
- الوصف
- 707-723 p.
- الموضوعات
- Amyotrophic Lateral Sclerosis؛ Animals؛ Drosophila؛ Gene Knock-In Techniques؛ Humans؛ Locomotion؛ Longevity؛ Motor Neurons