Human SOD1 ALS Mutations in a Drosophila Knock-In Model Cause Severe Phenotypes and Reveal Dosage-Sensitive Gain- and Loss-of-Function Components.

Human SOD1 ALS Mutations in a Drosophila Knock-In Model Cause Severe Phenotypes and Reveal Dosage-Sensitive Gain- and Loss-of-Function Components. — المؤلف: Şahin, Aslı — Genetics 20170530

المؤلف
Şahin, Aslı
النشر
Genetics 20170530
الوصف
707-723 p.
الموضوعات
Amyotrophic Lateral Sclerosis؛ Animals؛ Drosophila؛ Gene Knock-In Techniques؛ Humans؛ Locomotion؛ Longevity؛ Motor Neurons