A hyperprostaglandin E syndrome mutation in Kir1.1 (renal outer medullary potassium) channels reveals a crucial residue for channel function in Kir1.3 channels. [electronic resource]
Producer: 19981013Description: 23884-91 p. digitalISSN:- 0021-9258
- Amino Acid Sequence
- Animals
- Bartter Syndrome -- embryology
- COS Cells
- Cloning, Molecular
- Female
- Humans
- Kidney Medulla -- metabolism
- Molecular Sequence Data
- Mutagenesis, Site-Directed
- Oocytes -- physiology
- Point Mutation
- Polymerase Chain Reaction
- Polymorphism, Single-Stranded Conformational
- Potassium Channels -- chemistry
- Potassium Channels, Inwardly Rectifying
- Prostaglandins E -- metabolism
- Recombinant Proteins -- chemistry
- Sequence Alignment
- Sequence Homology, Amino Acid
- Syndrome
- Transfection
- Xenopus laevis
- Kcnj10 Channel
No physical items for this record
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
There are no comments on this title.
Log in to your account to post a comment.