18α-Glycyrrhetinic acid (GA) ameliorates fructose-induced nephropathy in mice by suppressing oxidative stress, dyslipidemia and inflammation. [electronic resource]
Producer: 20210119Description: 109702 p. digitalISSN:- 1950-6007
- Acute Kidney Injury -- chemically induced
- Animals
- Anti-Inflammatory Agents -- pharmacology
- Cell Survival -- drug effects
- Cytokines -- metabolism
- Dyslipidemias -- metabolism
- Fatty Acids -- metabolism
- Fructose -- pharmacology
- Glycyrrhetinic Acid -- analogs & derivatives
- Glycyrrhiza -- chemistry
- Humans
- Inflammation -- metabolism
- MAP Kinase Signaling System -- drug effects
- Male
- Mice
- Mice, Inbred C57BL
- Mitochondria -- pathology
- NF-E2-Related Factor 2 -- metabolism
- NF-kappa B -- metabolism
- Oxidative Stress -- drug effects
No physical items for this record
Publication Type: Journal Article
There are no comments on this title.
Log in to your account to post a comment.