UCH-L1 bypasses mTOR to promote protein biosynthesis and is required for MYC-driven lymphomagenesis in mice. [electronic resource]
Producer: 20190716Description: 2564-2574 p. digitalISSN:- 1528-0020
- Animals
- Cell Transformation, Neoplastic -- genetics
- Lymphoma, B-Cell -- genetics
- Mechanistic Target of Rapamycin Complex 1 -- genetics
- Mechanistic Target of Rapamycin Complex 2 -- genetics
- Mice
- Mice, Knockout
- Neoplasm Proteins -- genetics
- Phosphorylation
- TOR Serine-Threonine Kinases -- genetics
- Ubiquitin Thiolesterase -- genetics
No physical items for this record
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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