Independent modes of disease repair by AIM protein distinguished in AIM-felinized mice. [electronic resource]
Producer: 20191025Description: 13157 p. digitalISSN:- 2045-2322
- Adipocytes -- immunology
- Animals
- Apoptosis Regulatory Proteins -- blood
- Carcinoma, Hepatocellular -- etiology
- Cats
- Diet, High-Fat -- adverse effects
- Disease Resistance -- genetics
- Fatty Liver -- etiology
- Gene Expression Regulation
- Hepatocytes -- immunology
- Immunoglobulin M -- blood
- Kupffer Cells -- immunology
- Liver Neoplasms -- etiology
- Mice
- Mice, Transgenic
- Obesity -- etiology
- Protein Binding
- Receptors, Immunologic -- blood
- Signal Transduction
- Transgenes
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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