Extracellular anti-angiogenic proteins augment an endosomal protein trafficking pathway to reach mitochondria and execute apoptosis in HUVECs. [electronic resource]
Producer: 20190905Description: 1905-1920 p. digitalISSN:- 1476-5403
- Angiostatins -- genetics
- Apoptosis -- drug effects
- Cell Membrane -- metabolism
- Endocytosis
- Endoplasmic Reticulum Chaperone BiP
- Endosomes -- metabolism
- Fibronectins -- pharmacology
- Fluorescence Recovery After Photobleaching
- Heat-Shock Proteins -- chemistry
- Human Umbilical Vein Endothelial Cells
- Humans
- Microscopy, Fluorescence
- Mitochondria -- metabolism
- Neovascularization, Physiologic -- drug effects
- Phosphoproteins -- antagonists & inhibitors
- Protein Transport -- drug effects
- RNA Interference
- RNA, Small Interfering -- metabolism
- Recombinant Proteins -- biosynthesis
- Sodium-Hydrogen Exchangers -- antagonists & inhibitors
- Synaptosomal-Associated Protein 25 -- antagonists & inhibitors
- Thrombospondins -- genetics
No physical items for this record
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
There are no comments on this title.
Log in to your account to post a comment.