Galangin ameliorates cisplatin-induced nephrotoxicity by attenuating oxidative stress, inflammation and cell death in mice through inhibition of ERK and NF-kappaB signaling. [electronic resource]
Producer: 20170731Description: 128-139 p. digitalISSN:- 1096-0333
- Acute Kidney Injury -- chemically induced
- Animals
- Anti-Inflammatory Agents -- pharmacology
- Antioxidants -- pharmacology
- Apoptosis -- drug effects
- Apoptosis Regulatory Proteins -- metabolism
- Cisplatin
- Cytokines -- metabolism
- Cytoprotection
- Disease Models, Animal
- Dose-Response Relationship, Drug
- Extracellular Signal-Regulated MAP Kinases -- metabolism
- Flavonoids -- pharmacology
- Glutamate-Cysteine Ligase -- metabolism
- Heme Oxygenase-1 -- metabolism
- Inflammation Mediators -- metabolism
- Kidney -- drug effects
- Male
- Malondialdehyde -- metabolism
- Membrane Proteins -- metabolism
- Mice, Inbred BALB C
- NF-E2-Related Factor 2 -- metabolism
- NF-kappa B -- metabolism
- Oxidative Stress -- drug effects
- Phosphorylation
- Signal Transduction -- drug effects
- Tyrosine -- analogs & derivatives
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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