Association of Serum HMGB2 Levels With In-Stent Restenosis: HMGB2 Promotes Neointimal Hyperplasia in Mice With Femoral Artery Injury and Proliferation and Migration of VSMCs. [electronic resource]
Producer: 20170621Description: 717-729 p. digitalISSN:- 1524-4636
- Aged
- Animals
- Biomarkers -- blood
- Case-Control Studies
- Cell Movement
- Cell Proliferation
- Cells, Cultured
- Coronary Angiography
- Coronary Artery Disease -- blood
- Coronary Restenosis -- blood
- Disease Models, Animal
- Female
- Femoral Artery -- injuries
- Genetic Predisposition to Disease
- HMGB2 Protein -- deficiency
- Humans
- Hyperplasia
- Logistic Models
- Male
- Mice, Inbred C57BL
- Mice, Knockout
- Middle Aged
- Multivariate Analysis
- Muscle, Smooth, Vascular -- injuries
- Myocytes, Smooth Muscle -- metabolism
- NADPH Oxidases -- metabolism
- Neointima
- Percutaneous Coronary Intervention -- adverse effects
- Phenotype
- Phosphorylation
- RNA Interference
- Reactive Oxygen Species -- metabolism
- Receptor for Advanced Glycation End Products -- genetics
- Risk Factors
- Signal Transduction
- Stents
- Swine
- Swine, Miniature
- Transfection
- Vascular System Injuries -- blood
No physical items for this record
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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