Blocking GluN2B subunits reverses the enhanced seizure susceptibility after prolonged febrile seizures with a wide therapeutic time-window. [electronic resource]
Producer: 20170505Description: 29-38 p. digitalISSN:- 1090-2430
- Animals
- Animals, Newborn
- Disease Models, Animal
- Disease Susceptibility -- metabolism
- Electroshock -- adverse effects
- Excitatory Amino Acid Agonists -- toxicity
- Excitatory Amino Acid Antagonists -- therapeutic use
- Exploratory Behavior -- drug effects
- Humans
- Interleukin 1 Receptor Antagonist Protein -- therapeutic use
- Interleukin-1beta -- therapeutic use
- Kainic Acid -- toxicity
- Maze Learning -- drug effects
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Piperidines -- therapeutic use
- Rats
- Rats, Sprague-Dawley
- Receptors, Interleukin-1 Type I -- genetics
- Receptors, N-Methyl-D-Aspartate -- metabolism
- Seizures, Febrile -- etiology
No physical items for this record
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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