APA

Newton K., Dugger D. L., Maltzman A., Greve J. M., Hedehus M., Martin-McNulty B., Carano R. A. D., Cao T. C., van Bruggen N., Bernstein L., Lee W. P., Wu X., DeVoss J., Zhang J., Jeet S., Peng I., McKenzie B. S., Roose-Girma M., Caplazi P., Diehl L., Webster J. D. & Vucic D. (20180116). RIPK3 deficiency or catalytically inactive RIPK1 provides greater benefit than MLKL deficiency in mouse models of inflammation and tissue injury. : Cell death and differentiation.

Chicago

Newton K, Dugger D L, Maltzman A, Greve J M, Hedehus M, Martin-McNulty B, Carano R A D, Cao T C, van Bruggen N, Bernstein L, Lee W P, Wu X, DeVoss J, Zhang J, Jeet S, Peng I, McKenzie B S, Roose-Girma M, Caplazi P, Diehl L, Webster J D and Vucic D. 20180116. RIPK3 deficiency or catalytically inactive RIPK1 provides greater benefit than MLKL deficiency in mouse models of inflammation and tissue injury. : Cell death and differentiation.

Harvard

Newton K., Dugger D. L., Maltzman A., Greve J. M., Hedehus M., Martin-McNulty B., Carano R. A. D., Cao T. C., van Bruggen N., Bernstein L., Lee W. P., Wu X., DeVoss J., Zhang J., Jeet S., Peng I., McKenzie B. S., Roose-Girma M., Caplazi P., Diehl L., Webster J. D. and Vucic D. (20180116). RIPK3 deficiency or catalytically inactive RIPK1 provides greater benefit than MLKL deficiency in mouse models of inflammation and tissue injury. : Cell death and differentiation.

MLA

Newton K, Dugger D L, Maltzman A, Greve J M, Hedehus M, Martin-McNulty B, Carano R A D, Cao T C, van Bruggen N, Bernstein L, Lee W P, Wu X, DeVoss J, Zhang J, Jeet S, Peng I, McKenzie B S, Roose-Girma M, Caplazi P, Diehl L, Webster J D and Vucic D. RIPK3 deficiency or catalytically inactive RIPK1 provides greater benefit than MLKL deficiency in mouse models of inflammation and tissue injury. : Cell death and differentiation. 20180116.