Mechanism of the induction of endoplasmic reticulum stress by the anti-cancer agent, di-2-pyridylketone 4,4-dimethyl-3-thiosemicarbazone (Dp44mT): Activation of PERK/eIF2α, IRE1α, ATF6 and calmodulin kinase. [electronic resource]
Producer: 20170426Description: 27-47 p. digitalISSN:- 1873-2968
- Activating Transcription Factor 6 -- agonists
- Animals
- Antineoplastic Agents -- chemical synthesis
- Apoptosis -- drug effects
- Calcium-Calmodulin-Dependent Protein Kinase Type 2 -- genetics
- Calcium-Calmodulin-Dependent Protein Kinases -- genetics
- Cell Line, Tumor
- Deferoxamine -- pharmacology
- Endoplasmic Reticulum -- drug effects
- Endoplasmic Reticulum Chaperone BiP
- Endoplasmic Reticulum Stress -- drug effects
- Endoribonucleases -- genetics
- Gene Expression Regulation, Neoplastic
- HSP40 Heat-Shock Proteins -- genetics
- Heat-Shock Proteins -- genetics
- Humans
- Iron Chelating Agents -- chemical synthesis
- Oxidation-Reduction
- Phosphorylation -- drug effects
- Protein Serine-Threonine Kinases -- genetics
- Signal Transduction
- Thiosemicarbazones -- chemical synthesis
- Transcription Factor CHOP -- genetics
- X-Box Binding Protein 1 -- genetics
- eIF-2 Kinase -- genetics
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Publication Type: Journal Article
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