Bit1 knockdown contributes to growth suppression as well as the decreases of migration and invasion abilities in esophageal squamous cell carcinoma via suppressing FAK-paxillin pathway. [electronic resource]
Producer: 20160930Description: 23 p. digitalISSN:- 1476-4598
- Animals
- Apoptosis
- Carboxylic Ester Hydrolases -- metabolism
- Carcinogenesis -- pathology
- Carcinoma, Squamous Cell -- pathology
- Cell Line, Tumor
- Cell Movement
- Cell Proliferation
- Down-Regulation
- Esophageal Neoplasms -- pathology
- Esophageal Squamous Cell Carcinoma
- Focal Adhesion Protein-Tyrosine Kinases -- metabolism
- Gene Knockdown Techniques
- Humans
- Matrix Metalloproteinase 2 -- metabolism
- Mice, Nude
- Mitochondrial Proteins -- metabolism
- Neoplasm Invasiveness
- Oligonucleotide Array Sequence Analysis
- Paxillin -- metabolism
- Protein Binding
- Proto-Oncogene Proteins c-bcl-2 -- metabolism
- RNA, Small Interfering -- metabolism
- Signal Transduction
- Xenograft Model Antitumor Assays
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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