Allyl-isatin suppresses cell viability, induces cell cycle arrest, and promotes cell apoptosis in hepatocellular carcinoma HepG2 cells. [electronic resource]
Producer: 20170406Description: 253-62 p. digitalISSN:- 1472-8206
- Antineoplastic Agents -- pharmacology
- Apoptosis -- drug effects
- Carcinoma, Hepatocellular -- drug therapy
- Cell Cycle Checkpoints -- drug effects
- Cell Survival -- drug effects
- Dose-Response Relationship, Drug
- Hep G2 Cells
- Humans
- Isatin -- pharmacology
- Liver Neoplasms -- drug therapy
- Monoamine Oxidase Inhibitors
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Publication Type: Journal Article
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