Ndufc2 Gene Inhibition Is Associated With Mitochondrial Dysfunction and Increased Stroke Susceptibility in an Animal Model of Complex Human Disease. [electronic resource]
Producer: 20170110ISSN:- 2047-9980
- Adenosine Triphosphate -- metabolism
- Adult
- Age of Onset
- Animals
- Brain -- enzymology
- Cell Line
- Chi-Square Distribution
- Databases, Genetic
- Disease Models, Animal
- Electron Transport Complex I -- deficiency
- Gene Deletion
- Gene Expression Profiling -- methods
- Gene Frequency
- Genetic Predisposition to Disease
- Heterozygote
- Humans
- Hypertension -- complications
- Logistic Models
- Male
- Membrane Potential, Mitochondrial
- Middle Aged
- Mitochondria -- enzymology
- Mitochondrial Diseases -- enzymology
- Multivariate Analysis
- Odds Ratio
- Oligonucleotide Array Sequence Analysis
- Phenotype
- Polymorphism, Single Nucleotide
- Quantitative Trait Loci
- RNA Interference
- Rats, Inbred SHR
- Rats, Transgenic
- Risk Factors
- Stroke -- enzymology
- Transfection
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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