Biocompatible arsenic trioxide nanoparticles induce cell cycle arrest by p21(WAF1/CIP1) expression via epigenetic remodeling in LNCaP and PC3 cell lines. [electronic resource]
Producer: 20160728Description: 41-52 p. digitalISSN:- 1879-0631
- Antineoplastic Agents -- pharmacology
- Arsenic Trioxide
- Arsenicals -- pharmacology
- Biocompatible Materials -- pharmacology
- Cell Cycle Checkpoints -- drug effects
- Cell Line, Tumor
- Cyclin-Dependent Kinase Inhibitor p21 -- biosynthesis
- Dose-Response Relationship, Drug
- Epigenesis, Genetic -- drug effects
- Humans
- Male
- Metal Nanoparticles -- therapeutic use
- Oxides -- pharmacology
- Prostatic Neoplasms -- drug therapy
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Publication Type: Journal Article
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