Tumor-selective use of DNA base excision repair inhibition in pancreatic cancer using the NQO1 bioactivatable drug, β-lapachone. [electronic resource]
Producer: 20160921Description: 17066 p. digitalISSN:- 2045-2322
- Animals
- Autophagy -- drug effects
- Catalase -- genetics
- Cell Line, Tumor
- Cell Survival -- drug effects
- DNA Breaks, Double-Stranded -- drug effects
- DNA Glycosylases -- antagonists & inhibitors
- DNA Repair -- drug effects
- DNA-Binding Proteins -- antagonists & inhibitors
- Dicumarol -- pharmacology
- Female
- Humans
- Hydroxylamines -- pharmacology
- Mice
- Mice, Nude
- NAD(P)H Dehydrogenase (Quinone) -- antagonists & inhibitors
- Naphthoquinones -- pharmacology
- Pancreatic Neoplasms -- drug therapy
- Poly(ADP-ribose) Polymerases -- metabolism
- Reactive Oxygen Species -- metabolism
- Transplantation, Heterologous
- X-ray Repair Cross Complementing Protein 1
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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