Interleukin-23 (IL-23), independent of IL-17 and IL-22, drives neutrophil recruitment and innate inflammation during Clostridium difficile colitis in mice. [electronic resource]
Producer: 20160531Description: 114-24 p. digitalISSN:- 1365-2567
- Animals
- Antibodies, Monoclonal -- administration & dosage
- Clostridioides difficile -- immunology
- Colon -- drug effects
- Disease Models, Animal
- Enterocolitis, Pseudomembranous -- immunology
- Female
- Immunity, Innate -- drug effects
- Inflammation Mediators -- immunology
- Interleukin-17 -- deficiency
- Interleukin-23 Subunit p19 -- deficiency
- Interleukins -- antagonists & inhibitors
- Intestinal Mucosa -- drug effects
- Male
- Mice, Inbred C57BL
- Mice, Knockout
- Neutrophil Infiltration -- drug effects
- Neutrophils -- drug effects
- Pancreatitis-Associated Proteins
- Proteins -- immunology
- Signal Transduction
- Interleukin-22
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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