IL-21R signaling is critical for induction of spontaneous experimental autoimmune encephalomyelitis. [electronic resource]
Producer: 20160222Description: 4011-20 p. digitalISSN:- 1558-8238
- Animals
- Antigen Presentation
- Cells, Cultured
- Disease Susceptibility
- Encephalomyelitis, Autoimmune, Experimental -- immunology
- Interferon-gamma -- biosynthesis
- Interleukin-21 Receptor alpha Subunit -- deficiency
- Interleukins -- physiology
- Lymphocyte Activation
- Lymphopoiesis
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Mice, Transgenic
- Myelin-Oligodendrocyte Glycoprotein -- immunology
- Peptide Fragments -- immunology
- Receptors, Interleukin -- biosynthesis
- Signal Transduction
- Specific Pathogen-Free Organisms
- Th17 Cells -- immunology
- Interleukin-21
No physical items for this record
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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