Inhibition of microglial activation contributes to propofol-induced protection against post-cardiac arrest brain injury in rats. [electronic resource]
Producer: 20151103Description: 892-903 p. digitalISSN:- 1471-4159
- Adenosine Triphosphate -- physiology
- Animals
- Asphyxia -- complications
- Brain Ischemia -- etiology
- CA1 Region, Hippocampal -- drug effects
- CD11b Antigen -- biosynthesis
- Cardiopulmonary Resuscitation
- Cells, Cultured
- Heart Arrest -- complications
- Interleukin-1beta -- metabolism
- MAP Kinase Signaling System -- drug effects
- Male
- Maze Learning
- Mice
- Microglia -- drug effects
- Minocycline -- pharmacology
- Models, Animal
- Nerve Tissue Proteins -- biosynthesis
- Neuroprotective Agents -- pharmacology
- Phosphorylation
- Propofol -- pharmacology
- Protein Processing, Post-Translational
- Random Allocation
- Rats
- Rats, Sprague-Dawley
- Receptors, Purinergic P2X7 -- biosynthesis
- Tumor Necrosis Factor-alpha -- metabolism
- p38 Mitogen-Activated Protein Kinases -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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