NOX1 is responsible for cell death through STAT3 activation in hyperoxia and is associated with the pathogenesis of acute respiratory distress syndrome. [electronic resource]
Producer: 20141014Description: 537-51 p. digitalISSN:- 1936-2625
- Animals
- Case-Control Studies
- Caspase 3 -- metabolism
- Cell Death
- Cells, Cultured
- Disease Models, Animal
- Epithelial Cells -- enzymology
- Female
- Humans
- Hyperoxia -- enzymology
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- NADH, NADPH Oxidoreductases -- deficiency
- NADPH Oxidase 1
- NADPH Oxidases -- metabolism
- Phosphorylation
- Poly (ADP-Ribose) Polymerase-1
- Poly(ADP-ribose) Polymerases -- metabolism
- Pulmonary Alveoli -- enzymology
- RNA Interference
- Reactive Oxygen Species -- metabolism
- Respiratory Distress Syndrome -- enzymology
- STAT3 Transcription Factor -- metabolism
- Signal Transduction
- Time Factors
- Transfection
No physical items for this record
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
There are no comments on this title.
Log in to your account to post a comment.