TGF-β1 stimulates mitochondrial oxidative phosphorylation and generation of reactive oxygen species in cultured mouse podocytes, mediated in part by the mTOR pathway. [electronic resource]
Producer: 20140116Description: F1477-90 p. digitalISSN:- 1522-1466
- Adenosine Triphosphate -- metabolism
- Animals
- Antigens, Polyomavirus Transforming -- genetics
- Apoptosis -- drug effects
- Biosensing Techniques
- Cells, Cultured
- Dose-Response Relationship, Drug
- Fatty Acids -- metabolism
- Glycolysis -- drug effects
- Humans
- Hydrogen-Ion Concentration
- Intracellular Signaling Peptides and Proteins -- genetics
- Membrane Potential, Mitochondrial -- drug effects
- Membrane Proteins -- genetics
- Mice
- Mice, Transgenic
- Mitochondria -- drug effects
- Oxidative Phosphorylation -- drug effects
- Podocytes -- drug effects
- Protein Kinase Inhibitors -- pharmacology
- Reactive Oxygen Species -- metabolism
- Recombinant Proteins -- pharmacology
- Signal Transduction -- drug effects
- TOR Serine-Threonine Kinases -- antagonists & inhibitors
- Time Factors
- Transforming Growth Factor beta1 -- pharmacology
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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