eNOS deficiency predisposes podocytes to injury in diabetes. [electronic resource]
Producer: 20130110Description: 1810-23 p. digitalISSN:- 1533-3450
- Albuminuria -- etiology
- Angiotensin-Converting Enzyme Inhibitors -- pharmacology
- Animals
- Capillaries -- pathology
- Diabetes Mellitus, Experimental -- metabolism
- Diabetic Nephropathies -- etiology
- Disease Models, Animal
- Glucose -- metabolism
- Humans
- Kidney Glomerulus -- blood supply
- Male
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Nitric Oxide Synthase Type III -- deficiency
- Podocytes -- drug effects
- Renin-Angiotensin System -- drug effects
- Vascular Endothelial Growth Factor Receptor-2 -- antagonists & inhibitors
- rho GTP-Binding Proteins -- metabolism
- rhoA GTP-Binding Protein
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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