Sodium channel Na(v)1.7 is essential for lowering heat pain threshold after burn injury. [electronic resource]
Producer: 20121026Description: 10819-32 p. digitalISSN:- 1529-2401
- Activating Transcription Factor 3 -- metabolism
- Analysis of Variance
- Animals
- Biophysics
- Burns -- complications
- Calcitonin Gene-Related Peptide -- metabolism
- Calcium -- metabolism
- Cells, Cultured
- Cytokines -- metabolism
- Disease Models, Animal
- Edema -- etiology
- Electric Stimulation
- Functional Laterality
- Ganglia, Spinal -- cytology
- Glycoproteins -- metabolism
- Hot Temperature -- adverse effects
- Hyperalgesia -- etiology
- Membrane Potentials -- drug effects
- Mice
- Mice, Inbred C57BL
- Mice, Transgenic
- NAV1.7 Voltage-Gated Sodium Channel
- NAV1.8 Voltage-Gated Sodium Channel
- NAV1.9 Voltage-Gated Sodium Channel
- Neuralgia -- etiology
- Neuroglia -- pathology
- Pain Threshold -- physiology
- Patch-Clamp Techniques
- Proteins -- genetics
- RNA, Messenger -- metabolism
- RNA, Untranslated
- Sensory Receptor Cells -- drug effects
- Sodium Channel Blockers -- pharmacology
- Sodium Channels -- deficiency
- Tetrodotoxin -- pharmacology
- Transfection
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Publication Type: Journal Article; Research Support, U.S. Gov't, Non-P.H.S.
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