Increased EID1 nuclear translocation impairs synaptic plasticity and memory function associated with pathogenesis of Alzheimer's disease. [electronic resource]
Producer: 20120607Description: 902-12 p. digitalISSN:- 1095-953X
- Alzheimer Disease -- complications
- Animals
- Brain -- anatomy & histology
- Cell Cycle Proteins
- Cell Line, Tumor
- Cell Nucleus -- pathology
- Electric Stimulation
- Glial Fibrillary Acidic Protein -- metabolism
- Histones -- metabolism
- Humans
- In Vitro Techniques
- Maze Learning -- physiology
- Membrane Potentials -- drug effects
- Membrane Proteins -- metabolism
- Memory Disorders -- etiology
- Mice
- Mice, Transgenic
- Microtubule-Associated Proteins -- metabolism
- Neuronal Plasticity -- physiology
- Neurons -- pathology
- Nuclear Proteins -- genetics
- Patch-Clamp Techniques
- Phosphoproteins -- metabolism
- Phosphopyruvate Hydratase -- metabolism
- Protein Transport -- physiology
- Repressor Proteins -- genetics
- Teratocarcinoma -- pathology
- Transfection
- Tumor Suppressor Protein p53 -- metabolism
- p300-CBP Transcription Factors -- metabolism
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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