Loss of nuclear pro-IL-16 facilitates cell cycle progression in human cutaneous T cell lymphoma. [electronic resource]
Producer: 20120131Description: 4838-49 p. digitalISSN:- 1558-8238
- Active Transport, Cell Nucleus
- Aged
- Aged, 80 and over
- Amino Acid Sequence
- CD4-Positive T-Lymphocytes -- metabolism
- Cell Cycle -- genetics
- Cyclin-Dependent Kinase Inhibitor p21 -- biosynthesis
- Cyclin-Dependent Kinase Inhibitor p27 -- biosynthesis
- DNA, Neoplasm -- genetics
- Female
- Gene Expression Regulation, Neoplastic
- HSC70 Heat-Shock Proteins -- physiology
- Humans
- Interleukin-16 -- genetics
- Lymphoma, T-Cell, Cutaneous -- genetics
- Male
- Middle Aged
- Molecular Sequence Data
- Mutation
- Neoplasm Proteins -- genetics
- Precursor T-Cell Lymphoblastic Leukemia-Lymphoma -- pathology
- Protein Precursors -- genetics
- Protein Structure, Tertiary
- S-Phase Kinase-Associated Proteins -- biosynthesis
- Sequence Alignment
- Sequence Analysis, DNA
- Sequence Analysis, Protein
- Sequence Homology, Amino Acid
- Sezary Syndrome -- genetics
- Skin Neoplasms -- genetics
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Publication Type: Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Retracted Publication
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