PI3Kγ is required for NMDA receptor-dependent long-term depression and behavioral flexibility. [electronic resource]
Producer: 20111221Description: 1447-54 p. digitalISSN:- 1546-1726
- Analysis of Variance
- Animals
- Behavior, Animal -- drug effects
- Biophysics
- Chromones -- pharmacology
- Class Ib Phosphatidylinositol 3-Kinase -- deficiency
- Conditioning, Classical -- drug effects
- Electric Stimulation -- methods
- Environment
- Enzyme Inhibitors -- pharmacology
- Excitatory Amino Acid Agents -- pharmacology
- Excitatory Postsynaptic Potentials -- drug effects
- Exploratory Behavior -- drug effects
- Extinction, Psychological -- physiology
- Fear -- physiology
- Female
- Gene Expression Regulation -- drug effects
- Glycogen Synthase Kinase 3 -- metabolism
- Glycogen Synthase Kinase 3 beta
- Hippocampus -- cytology
- In Vitro Techniques
- Long-Term Synaptic Depression -- drug effects
- Male
- Maze Learning -- drug effects
- Methoxyhydroxyphenylglycol -- analogs & derivatives
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Morpholines -- pharmacology
- Neurons -- drug effects
- Oncogene Protein v-akt -- genetics
- Phosphorylation -- genetics
- Quinoxalines -- pharmacology
- RNA, Messenger -- metabolism
- Receptors, N-Methyl-D-Aspartate -- metabolism
- Signal Transduction -- drug effects
- Thiazolidinediones -- pharmacology
- Time Factors
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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