IKBKE protein activates Akt independent of phosphatidylinositol 3-kinase/PDK1/mTORC2 and the pleckstrin homology domain to sustain malignant transformation. [electronic resource]
Producer: 20111215Description: 37389-98 p. digitalISSN:- 1083-351X
- 3-Phosphoinositide-Dependent Protein Kinases
- Animals
- Breast Neoplasms -- genetics
- Cell Line, Tumor
- Cell Transformation, Neoplastic -- genetics
- Enzyme Activation -- drug effects
- Female
- Gene Deletion
- HEK293 Cells
- Heterocyclic Compounds, 3-Ring -- pharmacology
- Humans
- I-kappa B Kinase -- genetics
- Mammary Neoplasms, Animal -- genetics
- Mice
- Mice, Knockout
- NIH 3T3 Cells
- Oxadiazoles -- pharmacology
- Phosphatidylinositol 3-Kinases -- genetics
- Phosphorylation -- drug effects
- Phosphorylcholine -- analogs & derivatives
- Protein Serine-Threonine Kinases -- genetics
- Protein Structure, Tertiary
- Proto-Oncogene Proteins c-akt -- antagonists & inhibitors
- Ribonucleosides -- pharmacology
- Trans-Activators -- genetics
- Transcription Factors -- genetics
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Retracted Publication
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