The histone trimethyllysine demethylase JMJD2A promotes cardiac hypertrophy in response to hypertrophic stimuli in mice. [electronic resource]
Producer: 20110817Description: 2447-56 p. digitalISSN:- 1558-8238
- Animals
- Aortic Diseases -- complications
- Base Sequence
- Cells, Cultured -- drug effects
- Constriction, Pathologic -- complications
- Female
- Gene Expression Regulation
- Heart Failure -- enzymology
- Histone Demethylases -- deficiency
- Histones -- metabolism
- Humans
- Hypertrophy, Left Ventricular -- enzymology
- Intracellular Signaling Peptides and Proteins
- Jumonji Domain-Containing Histone Demethylases -- genetics
- LIM Domain Proteins
- Male
- Methylation
- Mice
- Mice, Inbred C57BL
- Mice, Transgenic
- Molecular Sequence Data
- Muscle Proteins -- biosynthesis
- Myocytes, Cardiac -- drug effects
- Nuclear Proteins -- physiology
- Promoter Regions, Genetic
- Protein Processing, Post-Translational
- RNA, Small Interfering -- pharmacology
- Rats
- Serum Response Element
- Trans-Activators -- physiology
- Ventricular Remodeling -- genetics
No physical items for this record
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
There are no comments on this title.
Log in to your account to post a comment.