Cellular prion protein promotes glucose uptake through the Fyn-HIF-2α-Glut1 pathway to support colorectal cancer cell survival. [electronic resource]

By:

Li, Qing-Quan

Contributor(s):

Sun, Yan-Ping
Ruan, Can-Ping
Xu, Xin-Yun
Ge, Jun-Hui
He, Jin
Xu, Zu-De
Wang, Qiang
Gao, Wen-Chao

Producer: 20110311Description: 400-6 p. digitalISSN:

1349-7006

Subject(s):

Adenocarcinoma -- metabolism
Animals
Basic Helix-Loop-Helix Transcription Factors -- metabolism
Cell Survival
Chromatin Immunoprecipitation
Colorectal Neoplasms -- metabolism
Female
Glucose -- metabolism
Glucose Transporter Type 1 -- metabolism
Humans
Immunoblotting
Mice
Mice, Nude
Microscopy, Fluorescence
Polymerase Chain Reaction
PrPC Proteins -- metabolism
Proto-Oncogene Proteins c-fyn -- metabolism
Signal Transduction -- physiology
Xenograft Model Antitumor Assays

Online resources:

Available from publisher's website

In: Cancer science vol. 102

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Cellular prion protein promotes glucose uptake through the Fyn-HIF-2α-Glut1 pathway to support colorectal cancer cell survival.

APA

Li Q., Sun Y., Ruan C., Xu X., Ge J., He J., Xu Z., Wang Q. & Gao W. (20110311). Cellular prion protein promotes glucose uptake through the Fyn-HIF-2α-Glut1 pathway to support colorectal cancer cell survival. : Cancer science.

Chicago

Li Qing-Quan, Sun Yan-Ping, Ruan Can-Ping, Xu Xin-Yun, Ge Jun-Hui, He Jin, Xu Zu-De, Wang Qiang and Gao Wen-Chao. 20110311. Cellular prion protein promotes glucose uptake through the Fyn-HIF-2α-Glut1 pathway to support colorectal cancer cell survival. : Cancer science.

Harvard

Li Q., Sun Y., Ruan C., Xu X., Ge J., He J., Xu Z., Wang Q. and Gao W. (20110311). Cellular prion protein promotes glucose uptake through the Fyn-HIF-2α-Glut1 pathway to support colorectal cancer cell survival. : Cancer science.

MLA

Li Qing-Quan, Sun Yan-Ping, Ruan Can-Ping, Xu Xin-Yun, Ge Jun-Hui, He Jin, Xu Zu-De, Wang Qiang and Gao Wen-Chao. Cellular prion protein promotes glucose uptake through the Fyn-HIF-2α-Glut1 pathway to support colorectal cancer cell survival. : Cancer science. 20110311.

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