Tumor necrosis factor induces matrix metalloproteinases in cardiomyocytes and cardiofibroblasts differentially via superoxide production in a PI3Kgamma-dependent manner. [electronic resource]
Producer: 20100401Description: C679-92 p. digitalISSN:- 1522-1563
- Acetophenones -- pharmacology
- Animals
- Animals, Newborn
- Cardiomyopathies -- enzymology
- Cells, Cultured
- Class Ib Phosphatidylinositol 3-Kinase
- Culture Media, Conditioned -- metabolism
- Disease Models, Animal
- Enzyme Activation
- Enzyme Inhibitors -- pharmacology
- Fibroblasts -- drug effects
- Humans
- Isoenzymes -- deficiency
- Male
- Matrix Metalloproteinases -- metabolism
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Myocytes, Cardiac -- drug effects
- NADPH Oxidases -- antagonists & inhibitors
- Phosphatidylinositol 3-Kinases -- deficiency
- Recombinant Proteins -- metabolism
- Signal Transduction
- Superoxides -- metabolism
- Time Factors
- Tumor Necrosis Factor-alpha -- deficiency
- Up-Regulation
- Ventricular Remodeling
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Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
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